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The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Disruptions in mammary stem cell division can lead to cancer, but targeting these processes might help treat breast cancer.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

Injury boosts normal skin cell growth, reducing cancer cell advantage.

A gene mutation in mice causes permanent hair loss and skin issues.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

A new mutation in the PLEC gene causes a rare condition with skin blistering, muscle weakness, and hair loss.

Translation levels actively determine keratinocyte cell fate.

The Thr1022Ala variant in the hairless gene is not a disease-causing mutation.

Runx1 and CDK inhibitors help keep hair follicle stem cells inactive, affecting their future roles.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

ALRN-6924 may prevent hair loss caused by chemotherapy.

Myc changes chromatin in stem cells, causing them to leave their niche.

CEP135 may predict cancer outcomes, and targeting PLK1 could help treat certain sarcomas.

Human melanocytes have unique traits that affect melanoma development and prognosis.

Hair loss in certain mice is linked to changes in keratin-related genes.

Certain interactions help prepare the androgen receptor for pairing and activation, which is important for its role in development and disease.

The type of tumor suppressor affects the form of skin cancer from hair follicle stem cells.

EZH1 and EZH2 are crucial for healthy hair growth and skin repair.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

ETS2 is crucial in squamous cell carcinoma development and could be a therapeutic target.

Temporary activation of β-catenin can create new hair follicles, but ongoing activation is needed to keep hair follicle tumors.

Mature cells can re-enter the cell cycle and potentially lead to cancer.