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The study showed that a specific DNA sequence can control gene expression in hair growth areas of mice.

The LEF-1 gene in cashmere goats was successfully cloned and analyzed, showing potential for improving cashmere production.

The scraggly mutation causes hair loss and skin defects in mice.

m6A-circHECA may affect cashmere goat hair growth and is possibly controlled by gene promoter methylation.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Cathepsin E is crucial for normal skin cell differentiation and development.

Deleting the Sox21 gene changes hair lipid composition and increases cholesterol sulfate levels.

Hair root analysis can effectively detect somatic mosaicism in double cortex syndrome.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

Arginine converts to citrulline in hair follicles as proteins harden.

A rare case of isolated trichothiodystrophy was identified, marked by brittle hair with low sulfur content.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

CTCF protein is essential for skin and hair follicle development in mice.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Matrix effects in LC-MS can be managed but not completely avoided.

Msx2-deficient mice experience irregular hair growth and loss due to disrupted hair cycle phases.

Hair and wool have complex microscopic structures with microfibrils and varying cystine content.

A mutation in the KRT86 gene causes hair fragility in a Turkish family.

Two new gene mutations cause a rare hair disorder.

Inherited color dilution in rabbits is linked to DNA methylation changes.

Certain SPINK5 gene mutations are common in Israeli families with Comèl-Netherton syndrome.

New hair perming method using tyrosine is gentler and can be done with a blow-dryer.

A specific gene mutation causes a severe skin disorder in a family.

Mutants of CYP154C2 enzyme significantly improved steroid conversion efficiency.

Two patients had a unique form of trichothiodystrophy with reduced high-sulfur proteins in their hair.