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CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Researchers found a new genetic mutation linked to a hair condition in a Japanese boy.

Abnormal gene expression related to keratin causes hair loss in certain mice.

BMP signaling controls hair growth and skin color.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

The study maps how genes are regulated during mouse hair growth.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

Rac1 is essential for proper hair structure and color.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

Keratinocyte cytokines and genetic variations influence the development of moles and skin pigmentation.

Mouse zigzag hair bends form due to a 3-day cycle of changes in hair progenitors and their environment.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

GLI1 RNA CISH effectively identifies basal cell carcinoma but is less specific for benign follicular tumors.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

The study found markers indicating that cells responsible for hair color are differentiating in specific areas of the hair follicle.

Researchers found genes that may explain why some pigs grow winter hair, which could help breed cold-resistant pigs.

Recognizing minor skin lesions can help identify serious cancer syndromes.

K15 gene is mainly active in the basal layers of hair follicles and epithelia, aiding early skin cell development.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Hair patterns in mice are controlled by both a global system dependent on Fz6 and a local self-organizing system.

GLI and EGF signalling affect Basal Cell Carcinoma development and could be therapeutic targets.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

Lnc056 helps hair follicle stem cells grow by increasing TRIP6 expression.

DNMT3A is crucial for healthy skin and hair growth.