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CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Increased TEMRA cells can predict treatment outcomes in rapidly progressive alopecia areata.

Alopecia areata patients have more activated T cells in their blood, which may help in developing treatments.

Clonally expanded CD8+ T cells cause alopecia areata.

Amotosalen-treated donor T-cells can prevent late CMV infection after bone marrow transplants.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Developing human skin has immune cells with memory-like features.

Cells that move well may improve hair loss treatments by entering hair follicles.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

MITF+ melanoma patients are more likely to have multiple melanomas and unique skin patterns.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

Certain immune markers may predict chemotherapy response in mesothelioma, and nivolumab is a tolerable and effective treatment for advanced non-small cell lung cancer.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Targeting Vδ1+T-cells may help treat alopecia areata.

The skin can independently form immune responses through special structures, offering new ways to treat skin diseases.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

iNKT cells can help prevent and treat alopecia areata by promoting hair regrowth.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Lipid-antigen stimulation may play a role in folliculotropic mycosis fungoides.

People with acne have more CD4+ immune cells in their skin than healthy people.

Tph cells are linked to the severity of systemic lupus erythematosus.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Melanocytes might be targeted by the immune system in people with alopecia areata, but more research is needed.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.