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Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Some hair protein genes evolved early and were adapted for use in hair follicles.

Macrophage-stimulating protein helps hair grow and can start hair growth phase in mice and human hair samples.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Certain skin proteins can form anchoring structures without the protein AMACO.

Oncogenic K-ras causes rapid cancerous changes in the mouth's lining.

PBX1 reduces aging and cell death in stem cells by boosting SIRT1 and lowering PARP1.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

The KRTAP22-2 gene in sheep does not significantly affect wool traits.

AP-1 controls tumor cell type by affecting key signaling pathways.

LGR5 is a marker found in hair follicle stem cells in various species and is important for hair growth and skin repair.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

GPC1 is important for blood vessel growth in hair follicles and could help treat hair loss.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

A new syndrome was linked to two new genetic changes in the MBTPS1 gene in a 14-year-old girl.

c-Kit is important for heart regeneration and cancer development.

RAPK is a rare skin disorder with pigmented spots, mainly on hands and feet, starting in youth.

Diphencyprone initially increases mouse hair growth, then slows it, possibly due to changes in specific protein levels.

Phospholipase A2 enzymes play key roles in skin health and disease.

The MBTPS2 gene variant c.970+5G>A is a common mutation causing IFAP syndrome.

Mutations in the KRT85 gene cause hair and nail problems.

MITF+ melanoma patients are more likely to have multiple melanomas and unique skin patterns.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

The P133R mutation in AKR1D1 enzyme causes harmful bile acid buildup, explaining related health issues.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.