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ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

ROR2 is crucial for hair follicle stem cell maintenance and self-renewal.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

ROR2 is essential for hair follicle stem cell renewal and maintenance.

ROR-alpha may increase the growth of certain breast cancer cells by boosting aromatase, which could affect breast cancer prognosis.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

Antizyme slows skin tumor growth by reducing cell growth in mice.

Regulatory T cells enhance bone formation by influencing cell mechanics.

MPA increases cancer spread by boosting Eph A2 activity.

Androgens increase muscle mass by promoting myoblast growth through ornithine decarboxylase.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

ETC-159 was safe up to 30 mg, but no tumor shrinkage was seen.

Suppressing ODC activity reduces tumor growth in hair follicles.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

The mTOR signaling pathway might play a role in radiation dermatitis, but more research is needed.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

RORA plays a key role in controlling seasonal hair molting by affecting hair follicle cell activity.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Type 2 inflammation helps wound healing by switching immune cells to repair mode.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

RORA affects hair follicle stem cells' structure and movement, potentially helping treat hair loss.

Dihydrotestosterone (DHT) increases oil production in skin cells by activating mTOR, and mTOR inhibitors can reduce this effect.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Certain gene variations in AKT2 are more common in women with PCOS and are linked to higher levels of specific hormones and symptoms.