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Hair follicle stem cells can help treat ulcerative colitis in mice by releasing beneficial exosomes.

A new genetic disorder caused by an ODC1 mutation can be treated with DFMO.

The technique accurately identifies and evaluates hair follicle structures in skin.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

miR‑339‑5p can slow down hair follicle stem cell differentiation by targeting DLX5.

FOXN1 duplication can cause excessive hair growth.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

Mirvetuximab soravtansine improves quality of life and reduces symptoms more than standard chemotherapy in ovarian cancer patients.

Foxn1 is essential for hair pigmentation by directing pigment transfer to hair cells.

Mrp3 may aid in wound healing and hair growth.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

BMP5 is essential for ear cartilage cell growth in rodents.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

Slug (Snai2) helps regulate hair growth timing in mice.

Tigers had a skin condition causing hair loss and inflammation, but the cause is unknown and treatment didn't work.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

A specific molecular switch, driven by MAPK/ERK signaling, helps spiny mice heal wounds by regenerating skin instead of forming scars.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Matriptase imbalance contributes to cancer development and spread.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Some people with schwannomatosis have a new type of mutation in the LZTR1 gene.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Meis1 is crucial for skin health and tumor development.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.