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Generalized trichoepitheliomas with hair loss may indicate myasthenia gravis.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

A 29-year-old man had a jaw plaque diagnosed as follicular mucinosis, linked to nestin-positive hair follicle stem cells.

m6A-circHECA may affect cashmere goat hair growth and is possibly controlled by gene promoter methylation.

TRPV1 affects sebaceous gland function and could help treat acne.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

Androgens increase muscle mass by promoting myoblast growth through ornithine decarboxylase.

A patient with a new PLEC gene mutation showed symptoms of both muscular dystrophy and myasthenia gravis, which improved with steroid treatment.

Moracin M boosts hair growth by activating key pathways and promoting cell growth.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Myasthenia gravis can affect both motor and non-motor systems due to immune system issues.

Dopaminergic neurons in the gut have diverse subtypes with different neurotransmitter contents.

Certain skin cells near the base of hair muscles may help renew and stabilize skin, possibly affecting skin disorder understanding.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

Sex steroids influence skin appendage function and neoplasm development, with changes in AR and ERalpha in certain gland neoplasms.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

Trichoblastomas may mimic fetal skin development by having many Merkel cells, unlike adult skin.

A woman with myotonic dystrophy had multiple skin tumors on her scalp, suggesting a genetic link.

The mystacial pad's innervation in adult rats is more complex than previously thought.

Mutations in certain receptors can cause diseases and offer new treatment options.

Different ligands change the shape of the TRPV3 ion channel in unique ways.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

MC1R gene variations affect skin, hair color, UV sensitivity, and melanoma risk.