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Vitamin D receptor is important for regulating hair growth and wound healing in mice.

CTCF protein is essential for skin and hair follicle development in mice.

Vitamin D receptor helps prevent skin tumors.

Activating Kras in mouse skin causes excess skin and hair loss.

CD44 variant changes start alopecia areata, but don't maintain it.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Lower SMAD2/3 activation predicts more severe skin cancer.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

AZD6244 treatment causes skin aging effects by depleting skin stem cells.

A new DSG4 gene mutation causes hair defects in a young girl.

Stat3 activation increases hair follicle progenitors but reduces bulge region stem cells.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Stat3 affects skin cancer development by altering keratinocyte stem cell behavior.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

A new mutation in mice causes crooked whiskers and messy hair.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

ALX4 is crucial for normal craniofacial and hair development, with specific roles in different cell types.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

The Walleye dermal sarcoma virus cyclin causes excessive skin cell growth in mice.

Somatic BHD mutations are rare in Japanese renal tumors.

Different harmful mutations in the CDH3 gene cause HJMD, but symptoms vary among individuals.