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Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

c-Myc, Max, and Bin1 help hair follicle cells mature and die.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Methotrexate effectively controls eye inflammation in Japanese patients with non-infectious uveitis but requires careful monitoring for side effects.

Notch pathway activation causes abnormal hair layer development.

A new culture system can grow tooth-like structures from dental cells but can't yet develop roots.

Identical p53 gene mutations in different cancers suggest the need for careful treatment.

PRC1 is essential for proper skin development and stem cell formation by controlling gene activity.

Weak cell junctions disrupt hair follicle stem cell rest.

The cuneate nucleus has two main neuron types: relay neurons and interneurons.

Key genes and pathways influence wool traits in Merino sheep.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Polygonum multiflorum is used for health benefits but may cause liver issues.

A genetic mutation in the DCAF17 gene caused Woodhouse-Sakati syndrome in a Chinese patient from a related family.

A common mutation in the hHb6 gene is linked to monilethrix, but other factors may also play a role.

Disrupting β-catenin signaling in certain cells causes anorectal malformations.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

Specific mutations in a receptor cause facial abnormalities and hair loss.

Hair form in mixed-blood families varies due to hereditary twist-knots and pigment formation.

Early involution in Hirosaki hairless rats' mammary glands is linked to a unique modification of STAT5A.

Hairless protein helps control hair growth by regulating vitamin D receptor activity.

A new CDH3 gene mutation was found in a Spanish patient with sparse hair and eye issues.

Screening for the NUDT15 gene variant can prevent severe side effects from thiopurine drugs in East-Asian people.

CTCF protein is essential for skin and hair follicle development in mice.

Inherited ichthyoses cause widespread skin scaling and thickening due to gene mutations.