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Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

A new model for hair regeneration in mice was created in 2015, which is faster and less invasive than the old method, producing normal hairs in about 21 days.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

Higher PHGDH levels cause unusual melanin buildup in hair follicles.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

The HCR gene contributes to psoriasis risk.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

LGD1069 effectively prevents breast tumors in mice without toxicity.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Epidermal growth factor delays skin and hair development in mice.

Increased skin pigmentation in mice reduces bioluminescent signal accuracy.

Chronic T. gondii infection may harm male fertility.

Hair loss in certain mice is linked to changes in keratin-related genes.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Certain cells from hair follicles can create new hair and contribute to hair growth when implanted in mice.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Deleting Gpr54 speeds up hair growth and regeneration.

Mouse models help study genetic skin diseases.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

HGF/SF injections can stimulate hair growth and increase hair follicle size in mice.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Removing hair from mice without reproductive glands led to grey hair, possibly helping to understand greying in aging.

FGF signaling is crucial for starting feather development in chicken embryos.