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Finasteride use changes brain chemicals, causing lasting sexual issues and anxiety/depression.

Post-finasteride patients show changed neuroactive steroid levels, possibly causing erectile dysfunction and depression.

Finasteride treatment changes brain steroid levels and receptors, affecting brain function even after stopping treatment.

Gut microbes significantly affect brain steroid levels.

Stopping finasteride for male pattern hair loss changes neuroactive steroid levels in the brain and blood.

Finasteride affects brain and blood steroids, causing lasting sexual and emotional side effects.

Stress and alcohol affect brain chemicals differently in rats, mice, and humans, influenced by genetic differences.

Men and women have different levels and production of brain steroids, which may affect their risk for certain brain disorders.

Changing neuroactive steroid levels early in life can affect how adult rats respond to alcohol's stimulating effects.

Chronic ethanol increases certain brain receptor levels, influenced by steroids and protein changes.

Ethanol changes GABAA receptor α4 subunit levels through phosphorylation and neuroactive steroids.

Finasteride treatment in rats changed the expression of genes related to psychiatric and neurological functions, and these changes persisted after stopping the drug.

Depressed teens have different steroid levels in urine, which may help identify and treat them.

Finasteride treats enlarged prostate and hair loss, but may cause side effects in some patients.

Neuroactive steroids could become safe, effective treatments with more understanding of their complex brain actions and metabolism.

Neuroactive steroids may affect the risk and treatment of alcohol use disorders.

A new method accurately measures nine neuroactive steroids in small blood samples, helping to study brain diseases.

Short-term diabetes can cause changes in brain steroids, cholesterol balance, and mitochondrial function.

Stopping alcohol after long-term use causes anxiety and changes brain chemicals.

Early changes in brain chemicals affect how a drug reduces alcohol intake in rats.

Allopregnanolone may enhance alcohol's effects on dopamine neurons, influencing addiction risk.

Certain steroids in the brain affect mood and symptoms of depression, and treatments targeting these steroids show promise for improving these symptoms.

Early NAS level changes affect alcohol consumption vulnerability.

Inhibiting certain enzymes made female rats more sensitive to low-level pain.

Pregnancy increases certain GABA_A receptors in rat brains, influenced by steroid levels.

Blocking 5α-reductase can reduce sleep deprivation-related behavioral issues in rats.

Ethanol-induced motor incoordination in rats is not affected by increased neuroactive steroids.

Seeing trauma causes fear in mice by lowering their natural fear-reducing hormones.

Etifoxine, an anxiety drug, can lessen brain inflammation and cognitive issues in mice, partly by increasing production of protective brain steroids.

Finasteride treatment in male rats causes long-lasting effects on depression-like behavior, brain cell growth, inflammation, and gut bacteria composition.