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Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

PRP can improve hair growth in people with hair loss, but more research is needed.

Human hair follicle cells can be turned into neural stem cell-like cells, which might help treat brain diseases.

A new neural network helps identify key regulators in cell changes, aiding in understanding diseases and finding new treatments.

Gremlin proteins help regulate hair growth by balancing signals in hair follicles.

Neurosteroids help control dopamine responses in the brain.

Dopaminergic neurons in the gut have distinct subtypes, some releasing both dopamine and acetylcholine.

Lower growth factors linked to balding in androgenetic alopecia.

Certain mice without specific receptors or mast cells don't lose hair from stress.

Neurotrophins may contribute to genetic hair thinning by inhibiting hair growth.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Type II spiral ganglion neurites avoid high concentrations of laminin and fibronectin.

Dopaminergic neurons in the gut have diverse subtypes with different neurotransmitter contents.

Neurosteroids from glia cells help control seizure development in epilepsy.

miR-214-3p helps nerve repair and recovery.

miR-214-3p helps nerve repair and recovery.

OCT4 helps granulosa cell growth in early-stage follicles, and FSH increases OCT4 through specific pathways.

Higher levels of certain nerve-related proteins are linked to hair loss in women with scalp pain.

Progesterone protects rat brain cells by enhancing GABA A receptor activity.

Meis2 is essential for touch sensation and nerve function in mice.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Protease Nexin-1 is found in human hair growth cells and is affected by male hormones.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

CGRP-IR axons may help maintain and renew tissues.

Certain defective glucocorticoid receptor mutants move faster inside cell nuclei and work less effectively.