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HPV infection is linked to better survival in advanced anal cancer, higher radiation doses improve survival, especially in HPV-negative patients, and prostaglandin E₂ pretreatment can protect mouse hair follicles from radiation damage.

Keratin 17 is crucial for early hair strength and cell survival.

Deleting podoplanin in mice promotes hair growth by enhancing cell migration.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Patched1 helps prevent tumors by controlling cell growth.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

AP-1 controls tumor cell type by affecting key signaling pathways.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

p63 may influence skin cancer development and cell differentiation.

A new compound slows cancer cell growth and causes cell death by blocking cell cycle progression and increasing cell-damaging molecules.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

A truncated protein linked to breast cancer may change cell adhesion.

New treatments may restore cancer-blocking proteins, slow prostate cancer, identify drug targets, and potentially regrow hair.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

Krt16-deficient mice help understand skin disorders like PC and FNEPPK.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

HPV8 causes skin cancer by expanding specific skin stem cells.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

BMI1 is essential for preventing hair greying and maintaining hair color.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Artemis protein may help control hair growth and health by influencing cell processes.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.