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Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Activated ras can protect kidney cells from a certain substance that causes cell death.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Rac1 is crucial for normal hair structure and pigmentation.

A woman with a new PTCH gene mutation has both Gorlin syndrome and severe hair loss.

Blocking the Hedgehog pathway may help overcome drug resistance in multiple myeloma.

Targeting multiple pathways and understanding genetic mutations are crucial for effective melanoma therapy.

NBCCS and BFHS might be the same syndrome, helping better identify and manage cases.

New hair regrowth model introduced, imiquimod kills skin cancer cells, T-cadherin loss makes skin cancer more invasive, no strong link between PTCH1 gene and skin cancer after transplant, and male teens more likely to have hereditary hair loss.

p21waf1/cip1 and p27kip1 help in hair follicle differentiation in rats.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

GBP1 is a key target for treating Epstein-Barr virus-related kidney cancer, and finasteride may help.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

REV7 is crucial for genome stability and cancer treatment, making it a potential target for therapy.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

Breast cancer treatments work better with AR activation, improving results and reducing side effects.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

A new test helps find drugs to treat head and neck cancer by targeting c-Rel.

RACB in barley is crucial for cell polarity and nucleus positioning, aiding fungal infection.

Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

Wnt and Notch pathways are crucial for repairing blood stem cells after damage.

2-Methoxyestradiol causes cancer cell death by activating specific pathways, but androgens can block this effect.

Pachyonychia congenita is linked to a keratin gene on chromosome 17.

Changing YBX1 protein activity affects skin stem cell function and aging.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.