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HPV6/11 is often found in hair of men with anogenital warts, especially pubic hair, and can lead to recurrent warts.

Runx1 and CDK inhibitors help keep hair follicle stem cells inactive, affecting their future roles.

Different human hair follicle stem cells grow at different rates and respond differently to a baldness-related compound.

Loss of TET2 increases the risk of skin and oral cancer.

EGFR helps hair follicles transition properly by controlling Stathmin levels.

Prostaglandin helps regenerate hair follicles after radiation damage.

HOTTIP and miR-10b contribute to glioma therapy resistance by affecting cell behavior, suggesting they could be targets for treatment.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

Bald areas have lower cell growth, more DNA damage, and increased cell death.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

CD34 is crucial for skin tumor development in mice.

Mouse skin cells can become sperm-like cells in the lab.

Inhibiting PDE8A may help treat hair loss by boosting fat cell growth and hair regeneration.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

Integrin alpha-6 and p63 proteins may play a role in hair loss and are important for hair growth and maintenance.

Abnormal Hedgehog signaling in blood cancers may help tumors grow and resist chemotherapy, suggesting potential for targeted treatments.

Transplanted stem cells from hair follicles significantly boosted hair growth and normalized follicles in certain mice.

The analysis of a large pilomatricoma revealed five distinct areas with different gene activity related to hair growth and tumor development.

Stress in hair follicle stem cells causes inflammation in a chronic skin condition through a specific immune response pathway.

Chemotherapy improved a girl's painful foot condition linked to pachyonychia congenita.

A variant in the ADAM17 gene causes hair loss by increasing protein degradation through TRIM47.

A new genetic region, 17q21.31, is linked to higher ovarian cancer risk.

Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

The gene LRRC15 is more active in balding areas of the scalp compared to non-balding areas.

Activating telomerase can boost hair follicle stem cell growth and hair production.