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A specific gene mutation in Japanese people can cause varying degrees of hair thinning in adulthood.

Two specific SNPs in the TRPS1 gene cause excessive hair growth by altering the protein's structure.

A molecule called α-ketobutyrate was found to extend lifespan and improve aging-related symptoms in worms and mice by activating certain cellular pathways and may help develop anti-aging treatments for humans.

Suppressing ODC activity reduces tumor growth in hair follicles.

N-WASP is essential for normal hair growth in mice.

Glypican-1 is important for hair follicle blood vessel growth and could be a target for treating hair loss.

IL-1β inhibits human hair follicle growth.

The study showed that a specific DNA sequence can control gene expression in hair growth areas of mice.

A specific gene mutation causes a severe skin disorder in a family.

Fzd2 is important for skin and hair development through various signaling ways.

New mutations in the EBP gene cause CDPX2, affecting bones, skin, eyes, and hair, with females generally less affected than males.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

The most common sign of aging at the gene level is more Ectodysplasin A2 Receptor (EDA2R) being made.

A new genetic variant causes BRESHECK syndrome by disrupting cell growth and stress response.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The 190-kbp domain contains all human type I hair keratin genes, showing their organization and evolution.

H3K4me3 helps control RSPO3 to influence hair growth and development.

Sox2-expressing cells can help grow hair and heal skin.

The enzyme PA-PLA1α is important for proper hair follicle development.

Junctional proteins stabilize the inner root sheath and connect the companion layer in human hair.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Certain mice without specific receptors or mast cells don't lose hair from stress.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

Epimorphin helps start hair growth in mice.

KAP8.2 gene variations affect cashmere quality in goats.