Search

everythingexperts & clinicslearnresearchcommunity

for

Search:↓

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

PD‐L1 and PD‐L2 may not effectively control immune activation in alopecia areata.

PDGF signaling is crucial for cell development, wound healing, and fluid regulation in the body.

Urokinase, a type of protein, helps skin cells multiply faster, especially in newborn mice.

CLE40 and CRN/CLV2 pathways have opposite effects on root growth in Arabidopsis.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

OsPHR3 can help breed rice that efficiently uses phosphate without growth issues.

TCDD changes skin cell growth and keratin production in mice.

YAP1 is important for skin regeneration and may affect skin disorder treatments.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

ARQ-234, a protein designed to treat atopic dermatitis, shows increased effectiveness in early testing.

A new compound from Royoporus badius activates immune cells and induces inflammatory responses.

A substance called compound-1 could help increase hair growth by maintaining prostaglandin levels in hair follicles.

ROR-alpha may increase the growth of certain breast cancer cells by boosting aromatase, which could affect breast cancer prognosis.

Increased energy use in Pik3r1-related insulin resistance isn't due to Ucp1 thermogenesis.

Inhibiting TYK2 can restore hair growth in alopecia areata.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Mutations at Val-889 and Arg-752 disrupt key interactions in androgen receptor dimerization.

TMPRSS2 is crucial for COVID-19 infection and is a potential target for treatment.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Proper cell death regulation is crucial for normal hair follicle regeneration and skin remodeling.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

ATR is crucial for DNA repair and cell survival after UV exposure.

Scientists can control how skin stem cells divide by using different treatments.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

HPV8 causes hair follicle stem cells to grow, leading to skin lesions.