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A new compound slows cancer cell growth and causes cell death by blocking cell cycle progression and increasing cell-damaging molecules.

Carriers of a specific gene mutation have subtle skin changes without visible symptoms.

CDK4 levels affect the number of hair follicle stem cells in mice.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

The MAPK/ERK pathway is crucial in controlling cell growth and death in finasteride-induced hypospadias.

Dkk4 protein is necessary for the proper development and arrangement of hair follicles.

AMPK activation may reduce melanoma risk in red-haired individuals.

CircAGK affects cell death in hair cells by controlling the miR-3180-5p/BAX pathway, which can lead to hair loss.

ALRN-6924 may prevent hair loss caused by chemotherapy.

ATP-sensitive K+ channel subunits, particularly Sur2A, play a significant role in various cancers.

Delta1 is crucial for controlling skin cell growth and preventing tumors in mice.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

K10 may not prevent tumors as previously thought and might increase benign tumor risk.

CD2 might be a new treatment target for patchy alopecia areata.

The research linked PLCD1 gene variants to the development of trichilemmal cysts.

More PDCD4 is linked to obesity, insulin problems, and cell death in ovaries for those with polycystic ovary syndrome.

Upadacitinib effectively treats pyoderma gangrenosum.

PTCH gene mutations contribute to basal cell carcinoma development.

High glycine–tyrosine keratin-associated proteins help make hair strong and maintain its shape.

PRDX5 enzyme may contribute to alopecia areata by affecting oxidative stress and autoimmunity.

A new gene variant in the DSP gene is linked to a unique type of hair loss.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

Higher levels of the DP2 receptor may lead to hair loss.

A specific protein complex increases the activity of a plant enzyme, but this action is not required for plant root hair growth.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.