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A specific gene variant is linked to heart disease, increased heart muscle, curly hair, and thick skin on palms and soles.

aPKCλ is crucial for keeping hair follicle stem cells inactive and maintaining normal hair growth.

PBM helps improve cell survival in 3D tissue engineering.

Wnt signaling is essential for forming the skin's spinous layer through a BMP-FGF pathway.

The hedgehog signalling pathway is key in skin development and basal cell carcinoma, offering insights for prevention and treatment.

Mutations in the SNRPE gene cause hereditary hair loss.

MAA beads improved wound healing in male mice by activating the Shh pathway, but not in females.

Blocking SEPT4 might help heal wounds and regrow hair faster.

Kremen is crucial for proper development and preventing tumors by regulating Wnt signaling.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

The MBTPS2 gene variant c.970+5G>A is a common mutation causing IFAP syndrome.

SOX2 is crucial for skin cell function and hair growth, and it plays a role in skin cancer and wound healing.

Inhibiting SFRP1 may help treat hair loss.

The S1PR 1&4 modulator may effectively treat alopecia areata by reducing hair loss and immune cell activity.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

SM04554 may increase hair growth as a topical treatment for androgenetic alopecia.

Roots adapt to uneven environments by changing growth and gene expression.

Hairless protein is key for hair growth, cell differences cause gene expression variation, and the N-end rule pathway senses nitric oxide for protein breakdown.

Two specific genes are more active during hair growth in mice.

The study suggests that motor neurons created from stem cells of patients with spinal and bulbar muscular atrophy show signs of the disease, including changes in protein levels and cell functions.

miR-124 helps mouse hair follicle stem cells become nerve cells by blocking Ptbp1 and Sox9.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

HDAC4 and HDAC7 are crucial for Th17 cell development and could be targeted to treat inflammatory diseases.

LLPS is crucial for RALF signaling, aiding plant growth and stress resilience.

Slug is crucial for skin health, hair growth, and healing.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

Bone-forming cells grow well in 3D polymer scaffolds with 35 µm pores.

A new genetic mutation linked to CARASIL syndrome and small artery disease was found in a Chinese family.