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Mutations in specific genes cause certain congenital defects in dogs, aiding in understanding similar human diseases.

TG5 helps maintain hair follicle health, while TG3 aids in hair shaft development.

Controlling Tslp can improve health in AEC syndrome patients.

HPV8 causes skin cancer by expanding specific skin stem cells.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

The HPV type 11 region activates hair-specific gene expression in mice.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

TAZ boosts fat cell formation in goat stem cells by activating a specific signaling pathway.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

A new mutation in the HR gene causes hair loss in a specific family.

The Engrailed-1 gene plays a key role in the development of sweat glands in mice.

Atg5 can promote tumors when autophagy is deficient but suppresses them under normal conditions.

The Apc gene is crucial for normal skin and thymus development.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Removing anthrax toxin receptor 1 in pigs prevents Senecavirus A infection and causes a rare disease similar to GAPO syndrome.

Early over-expression of FoxN1 harms immune and skin development.

Gene therapy with the Math1 gene helped regenerate balance-related cells and improve balance in mice.

MCPIP1 in myeloid cells is important for skin cancer development and healthy hair growth.

Hereditary hyperplastic gingivitis in silver foxes may be linked to errors in the MAPK signaling pathway, influenced by androgens.

Overexpression of COX-2 in mice skin causes abnormal skin and hair development.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

P-cadherin is crucial for hair follicle pigmentation but not skin pigmentation.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Progesterone and its metabolites affect myelin protein expression differently in male and female rat Schwann cells.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

FOXN1 is crucial for thymus development and immune response in Xenopus laevis.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.