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Phosphorylation controls TFEB's location in the cell, affecting cell metabolism and stress response.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

cgVEGF164 boosts hair follicle growth in mice.

A five-month-old boy with Omenn syndrome successfully recovered after a stem cell transplant with reduced intensity conditioning.

Anti-TNF therapy can cause a rare hair loss condition similar to alopecia areata.

Trps1 plays a key role in hair follicle development and cycling.

Better DNA uptake in the skin leads to stronger immune responses.

A rare benign tumor with hair follicle features was found on a man's trunk.

Human thymus has stem cells that can self-renew and maintain their identity.

Reduced CD44 expression may cause abnormal hyaluronate buildup in cutaneous myxoma.

A 10-month-old boy with a rare combination of genetic conditions has severe immune deficiency and treatment challenges.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

Too much IKZF1 and Ikaros protein may cause alopecia areata.

Injecting certain cells into mice caused hair loss, which was preventable with a specific inhibitor.

Pemphigus vulgaris involves specific immune cells and B cells that produce antibodies causing skin blisters.

TGFβ-2 may cause hair loss in androgenetic alopecia.

A 7-year-old boy with a brain tumor developed early puberty, which was successfully treated with medication.

YAP1 is important for skin regeneration and may affect skin disorder treatments.

Researchers found a new genetic mutation linked to a hair condition in a Japanese boy.

The treatment showed significant hair regrowth in alopecia areata patients without side effects.

PCOS involves genetic and immune factors, especially T cells, affecting its development.

The photolyase-based device significantly changed the size and heat of potential skin cancer areas in patients.

Newly designed proteins can effectively degrade specific proteins in cells, offering a promising alternative for targeted protein degradation.

Different tumor cells in one basal cell carcinoma can cause mixed treatment responses, suggesting personalized treatment is needed.

Nonmelanoma skin cancers have higher levels of certain osteopontin variants than normal skin.

Trichoepitheliomas and some basal cell carcinomas likely come from hair follicle stem cells.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

A man developed autoimmune issues after a transplant, improved with treatment, but died from leukemia relapse.