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Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Smad2/3-dependent TGF-β signaling increases during wound healing.

Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

Wounded skin cells can revert to stem cells and help heal.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

The African spiny mouse heals skin without scarring due to different protein activity compared to the common house mouse, which heals with scarring.

Notch1 helps skin heal by attracting specific immune cells.

HMG-CoA reductase is crucial for skin wound healing by regulating keratinocyte growth and blood vessel formation.

Hair follicles help wounds heal faster, especially in active growth phase skin.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

A single PRP injection speeds up wound healing in both diabetic and non-diabetic rats.

African spiny mice can regenerate skin and hair after wounds due to specific tissue mechanics.

Keratin 6a is important for quick wound healing from hair follicles.

DPP4, a molecule in skin, helps heal large wounds and regrow hair follicles when its levels are reduced.

Glycogen levels in mouse skin drop after injury but increase during healing, returning to normal within a month.

PRP speeds up tongue wound healing in both diabetic and non-diabetic rats.

Thymosin β4 helps heal corneal wounds.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Myofibroblasts in rat wound healing may come from blood vessel pericytes and perifollicular dermal sheath cells.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Epigenetic changes are crucial for stem cell behavior in skin wound healing and their disruption may lead to cancer.

Bone marrow-derived stem cells greatly improve skin wound healing in rats.

House fly larvae substances improve wound healing and skin regeneration, especially in immunosuppressed mice.

Hair follicle stem cells help maintain skin health after injury.

Fibroblast growth factors are crucial for hair follicle development and regeneration.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Full thickness wounds on Lanyu pigs' skin resulted in abnormal skin structure and function due to changes in molecular expression patterns.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.