Search

everythinglearnresearchcommunity

for

Search:↓

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Lack of KSR1 stops certain skin tumors in mice.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Hair loss in certain mice is linked to changes in keratin-related genes.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

PP2Acα is essential for proper hair and skin development.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

The Bsk mutation doesn't involve keratin gene recombination and its cause is unknown.

Suppressing ODC activity reduces tumor growth in hair follicles.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Certain mice without specific receptors or mast cells don't lose hair from stress.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

The gene causing hair loss and heart issues in rough coat mice is still unknown.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Abnormal gene expression related to keratin causes hair loss in certain mice.

Using special RNA to target a mutant gene fixed hair problems in mice.

FoxN1 overexpression in young mice harms immune cell and skin development.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

Adult mice with CBS deficiency show minimal health issues and normal lifespan despite high homocysteine levels.

A chimeric keratin partially improved skin structure in mice lacking keratin 5, but didn't fully restore normal skin.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.