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Carriers of a specific gene mutation have subtle skin changes without visible symptoms.

A new laser technique can precisely remove specific cell types in living animals without harming nearby cells.

Inhibiting connective tissue sheath contraction may improve hair growth in male pattern baldness.

Antizyme slows skin tumor growth by reducing cell growth in mice.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

XEDAR triggers a specific signaling pathway in cells.

High glycine–tyrosine keratin-associated proteins help make hair strong and maintain its shape.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

848 genes related to fat and metabolism are less active in people with Central Centrifugal Cicatricial Alopecia.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

Blocking cell death in hair follicles can lead to impaired hair growth.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Genetic variants in specific genes cause central centrifugal cicatricial alopecia.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Increasing PDCD4 protein may help prevent or treat some skin cancers.

IL-19 and IL-24 help cells respond to DNA damage and could be targeted for cancer and age-related disease treatments.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Activating autophagy might reverse skin fibrosis.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

PROTACs offer new ways to treat hard-to-target diseases, with promising drugs for cancer in advanced trials.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

Skin tumors with CYLD cutaneous syndrome show more NF-κB activity and less organized collagen.

pCLCA2 protein may help maintain skin structure and function.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

The 4C32 gene may help in mouse skin development and differentiation.