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Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Msx2 deficiency in mice leads to bone growth and organ development problems.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

Ptprq has multiple forms that change during inner ear development.

Akt2 protein is essential for normal cell division in early mouse embryos.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

CTCF protein is essential for skin and hair follicle development in mice.

Trps1 is essential for proper hair follicle development.

Hair loss in certain mice is linked to changes in keratin-related genes.

Phospholipase C-δ1 is crucial for normal hair development.

CRABP1 boosts hair cell growth in Hu sheep by affecting key genes.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Pax9 is crucial for proper tongue surface development and preventing skin-like changes.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

The research cloned keratin 7 genes from humans, mice, and marsupials, found similarities between human and mouse genes, and discovered new areas of K7 expression in mice.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.