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Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Keratin 17 is crucial for early hair strength and cell survival.

Fatp4 is crucial for healthy skin development and function.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

The hairy ear mutation in mice is linked to changes in gene expression affecting hair growth.

A new keratin 6 type in mice explains why some mice without certain keratin genes still have normal hair and nails.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

A new mouse mutation causes skin and hair defects due to a gene change.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Akt2 protein is essential for normal cell division in early mouse embryos.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

A mutation in the HOXC13 gene causes hair and nail problems in a Syrian family.

Overexpressing PKCα in mice skin increases inflammation but doesn't affect tumor growth.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Loss of Msx2 function causes eye development issues similar to Peters anomaly.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.