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Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.

A new genetic mutation was found causing hair and eye issues in a boy.

The HCR gene contributes to psoriasis risk.

Aire mutation reduces alopecia areata, while Notch4 mutation prevents it in mice.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

A mouse model was created to study hair loss similar to humans.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Diphencyprone initially increases mouse hair growth, then slows it, possibly due to changes in specific protein levels.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

New mutations in the EBP gene cause CDPX2, affecting bones, skin, eyes, and hair, with females generally less affected than males.

Ptprq has multiple forms that change during inner ear development.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

Plet-1 protein helps hair follicle cells move and stick to tissues.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Impaired LEF1 activation speeds up skin cell development in Hutchinson-Gilford Progeria Syndrome.

Using special RNA to target a mutant gene fixed hair problems in mice.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

The scraggly mutation causes hair loss and skin defects in mice.

Enzyme replacement therapy improved multiple symptoms of homocystinuria in mice.

p120-catenin helps control skin inflammation by regulating cadherin levels.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

The naked mutation in mice causes hair loss and helps identify keratin genes.

Increased energy use in Pik3r1-related insulin resistance isn't due to Ucp1 thermogenesis.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Not having enough cystatin M/E protein causes less hair growth and dry skin.

Mutations in the HOXC13 gene cause hair and nail development issues.