Search

everythinglearnresearchcommunity

for

Search:↓

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

Stem cell niche exit in C. elegans is influenced by Sh1 cell membrane protrusions.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

HIV-1 may cause increased stem cell death in hair follicles, leading to hair loss.

Mitochondria change shape to meet energy needs during cell movement.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

Increased energy use in Pik3r1-related insulin resistance isn't due to Ucp1 thermogenesis.

Mitochondrial activity varies in cells before they stop growing, affecting their growth potential.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

Mice with enhanced regeneration abilities may help develop new regenerative medicine therapies.

Pallister-Killian Syndrome is a complex genetic disorder requiring coordinated care and genetic counseling.

Wnt/β-catenin signaling is crucial for cell fusion in placental development.

A girl with citrullinemia developed pili torti, suggesting a link between the hair condition and the metabolic disorder.

4-aminopyridine helps heal burn wounds faster by reducing inflammation and promoting new blood vessel growth.

A woman's hair grew back after treatment for a rare hair loss caused by proton therapy.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

DHA derivatives, especially didocosahexaenoin, effectively kill prostate cancer cells and may offer a safer cancer treatment option.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

Perhexiline can effectively target ovarian cancer cells left after treatment.

Activating TRPV3 channels stops hair growth by killing hair follicle cells.

Specific immune cells and pathways contribute to hair follicle inflammation and hair loss, suggesting potential treatments for lichen planopilaris.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Immune cells might contribute to hair loss caused by a specific mutation.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

Drugs targeting EMT molecules show promise for treating lichen planopilaris.

A new genetic mutation causing Xeroderma Pigmentosum was found in an 8-year-old girl, affecting her DNA repair.

Fatp4 is crucial for healthy skin development and function.

A man developed a rash similar to pityriasis rubra pilaris after starting sorafenib for cancer, possibly due to the drug's effect on skin cells.