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The enzyme RDH12 plays a role in vision and retinal disease, with mutations leading to early onset visual loss and blindness, but the exact disease mechanism is unclear and there are no treatments yet.

OsUEV1B protein is essential for controlling phosphate levels in rice.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

LPR1 regulates root growth under low phosphate stress independently of SIZ1 in Arabidopsis thaliana.

OsPHR3 can help breed rice that efficiently uses phosphate without growth issues.

A specific protein complex increases the activity of a plant enzyme, but this action is not required for plant root hair growth.

PADI4 enzyme slows down cell growth in developing hair follicles.

PTHrP regulates various normal body functions, including bone development, skin, and muscle function.

The P133R mutation in AKR1D1 enzyme causes harmful bile acid buildup, explaining related health issues.

Two new enzymes, PAD-R11 and PAD-R4, were cloned and showed activity, with PAD-R11 resembling epidermal enzymes.

LPAR6 has a unique way of binding and activating, which helps in designing treatments for hair loss and cancer.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Enzymes that change arginine to citrulline are important for skin barrier and hair formation, and their malfunction can lead to skin conditions and hair disorders.

PDF2 senses specific lipids and regulates root growth and gene expression in Arabidopsis.

This enzyme helps metabolize fatty acids and isoleucine, and could be key in treating neurological diseases and certain cancers.

The diphosphonate was the most effective at preventing calcification in rats.

The structure of SRD5A reveals how it reduces steroids, aiding drug design for related health conditions.

Increased energy use in Pik3r1-related insulin resistance isn't due to Ucp1 thermogenesis.

QSOX enzymes help form protein bonds in cells, especially in tissues with high secretory activity.

The protein's size was reduced, but more work is needed to confirm its function.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

The research explains how a human enzyme binds and processes its substrate, which could relate to its role in biological functions and hair loss.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

Activated ras can protect kidney cells from a certain substance that causes cell death.

Mutations in the enzyme don't significantly change how it binds to its specific substances.

A 9-year-old girl developed type 1 diabetes and alopecia after being treated for a rare kidney disease.