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A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

NFIC helps rat dental cells grow and turn into bone-like cells.

The mystacial pad's innervation in adult rats is more complex than previously thought.

CAR is a useful marker for assessing alopecia areata severity.

CRH promotes fat production in skin cells, affecting conditions like acne.

A reliable model for screening type II 5α-reductase inhibitors was created and validated.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

VEGF and VEGFR-2 genes influence feather maturity, and specific genetic markers can improve chicken breeding.

The gene causing hair loss and heart issues in rough coat mice is still unknown.

Rat hair follicle stem cells can be used to improve blood vessel growth in engineered skin.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

ESR2 gene linked to female-pattern hair loss.

Lack of TrkC receptor delays hair follicle development.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Astrotactin proteins are important for brain and skin development and are linked to several neurodevelopmental disorders.

A new compound was effective for imaging prostate cancer in rats.

Sacituzumab tirumotecan shows promise in treating breast cancer with manageable side effects.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Non-coding RNAs may be key in diagnosing and treating rare skin disorders.

TCDD reduces EGF receptors in the liver, affecting growth and development.

A new genetic mutation in the TRPS1 gene causes Trichorhinophalangeal Syndrome, leading to specific hair, dental, and bone issues.

Rat hair follicle stem cells can become corneal epithelium-like cells when exposed to rabbit corneal limbal stroma.

Dyclonine can effectively reduce skin issues by inhibiting the TRPV3 channel.

Inhibiting mTORC2 can reduce DNA repair and increase cancer cell death, suggesting potential for targeted brain cancer treatments.

Targeting Keratin 17 may help overcome cancer therapy resistance.

MOF controls skin development by regulating genes for mitochondria and cilia.

The treatment successfully reopened blood flow in most patients and extended the use of their dialysis access with low risk.