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Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

cgVEGF164 boosts hair follicle growth in mice.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

The N gene affects the protein makeup of mouse hair.

Aire mutation reduces alopecia areata, while Notch4 mutation prevents it in mice.

Mice with the naked gene have missing or abnormal hair cells.

A new mouse model effectively mimics vitiligo for research and drug testing.

Mouse models help study genetic skin diseases.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

A new mouse mutation causes skin and hair defects due to a gene change.

Retinoic acid affects male and female muscle energy use and function differently.

A mutation in the Sgkl gene causes defective hair growth in mice.

The mouse hairy ears mutation causes longer ear hair due to changes in gene expression.

Fgfr2b helps maintain healthy skin and prevent cancer.

A zinc-deficient diet stunted growth and affected organs in mice, with C57BL/6J mice showing more severe symptoms.

Rac1 is crucial for normal hair structure and pigmentation.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

The model effectively mimics radiation-induced skin damage for future research.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Prenatal retinoic acid exposure did not affect mouse vibrissal follicle development.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.