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A protein complex called mTORC1 likely affects when hair growth starts in mice.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Cyclosporin A and FK506 can start new hair growth without needing immunosuppression.

Rapamycin reduces age-related hearing cell loss in mice, but acarbose does not.

Topical rapamycin may effectively treat fibrous papules on the face.

The microneedle device with rapamycin and epigallocatechin gallate effectively promoted hair regrowth in mice.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.

Using an anti-ICAM-1 antibody with rapamycin improves hair transplant survival in monkeys.

Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Valproic acid and rapamycin protect hair follicle stem cells from damage by activating a protective pathway.

Angelica sinensis helps repair and regenerate the thymus in mice.

Rapamycin-primed exosomes can significantly boost hair regrowth.

Low-dose rapamycin microdepots can help regrow hair by activating certain cell processes.

A low dose of rapamycin increases inner ear hair cell creation by boosting SOX2+ cell numbers.

Rapamycin increased survival in mice with severe chronic graft-versus-host disease by expanding regulatory T cells.

Cyclosporin A and FK506 can start new hair growth in mice, but ascomycin and rapamycin cannot.

Eating a high-glycemic diet may worsen acne by increasing certain protein levels and expressions in the skin.

Acne patients have higher levels of mTOR in their skin, which could be linked to future metabolic disease.

mTORC2 is crucial for healthy skin barrier by regulating lipids and filaggrin.

mTOR may link different pathways in hair follicle tumor formation.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

The mTOR pathway may be involved in the development of hair follicle tumors, with higher activity in malignant tumors.

Acne patients have higher mTOR gene expression.

mTOR inhibitors may help treat lung fibrosis.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

The conclusion is that using drugs to block the TOR pathway might slow aging and prevent age-related diseases.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

Certain small molecules can promote hair growth by activating a cellular cleanup process called autophagy.

Overactive sonic hedgehog signaling worsens uterine scarring by reducing cell recycling.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.