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Aconiti Lateralis Preparata Radix helps mouse stem cells grow and turn into bone cells faster than usual methods.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

The study suggests that motor neurons created from stem cells of patients with spinal and bulbar muscular atrophy show signs of the disease, including changes in protein levels and cell functions.

Raptor and Rictor have stable expression in hair cycles, with Raptor marking stem cells and Rictor involved in hair shaft formation.

Scientists can control how skin stem cells divide by using different treatments.

Recognizing minor skin lesions can help identify serious cancer syndromes.

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Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

A new mutation in the KRT86 gene was found to cause the hair disorder monilethrix in a Han family.

The research identified and described a gene important for hormone conversion in endangered catfish, which varies in activity during different reproductive stages and after hormone treatment.

RCS-01 therapy is safe and may improve skin structure by affecting gene expression.

Kidney cancer cells without folliculin are more sensitive to radiation due to increased self-eating cell death.

Immune cells boost stem cell activity in hairy moles, causing more hair growth.

Small molecules KY19382 and KY19334 may help treat skin cancer by reducing CDK1 levels and blocking harmful cell signals.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

The mystacial pad's innervation in adult rats is more complex than previously thought.

Inherited color dilution in rabbits is linked to DNA methylation changes.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Mutations in Far2 mice cause hair loss due to sebaceous gland issues.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Keratin2-6g is crucial for hair follicle development, with mutations causing cell degeneration and vacuolation.

Peptide nanoparticles can effectively deliver CRISPR-Cas9 to target KRAS mutations in cancer.

The hairless gene mutation causes baldness by disrupting hair follicle structure.

Melanocytes are important for normal body functions and have potential uses in regenerative medicine and disease treatment.

Wnt and Notch pathways are crucial for repairing blood stem cells after damage.

The near-naked hairless mutation causes hair loss but is not due to a mutation in the hairless gene itself.

Gene expression affects fur development in rex rabbits.

Hair follicle stem cells can help treat ulcerative colitis in mice by releasing beneficial exosomes.

Removing Lrig1-positive cells in mice leads to temporary loss of sebaceous glands.