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All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Mice without certain skin proteins had abnormal skin and hair development.

GI198745 is more potent and longer-lasting than finasteride, potentially better for treating hair loss.

Topical minoxidil and tretinoin improved hair growth in a girl with EDSS1 over two years.

Retinoic acid reduces cell damage from oxidative stress.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

Double-stranded RNA helps regenerate hair follicles by increasing retinoic acid production and signaling.

We need to learn more about 5α-reductases and neuroactive steroids to safely make drugs targeting these enzymes.

Dietary L-serine supplementation can reduce symptoms of HSAN1 by lowering harmful 1-deoxySL levels.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

All-trans retinoic acid helps amelanotic melanocytes in hair follicles develop and produce pigment while reducing their growth.

Nocturnal calcium infusions improved a boy's severe rickets without causing hair loss.

Vitamin D receptor is crucial for bone health and mineral metabolism.

The gene HDC is important for the development of hair follicles in newborn mice.

5α-reductases increase epitestosterone's effect on androgen receptors.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

A PKP1 gene mutation causes skin fragility and hair loss in Chesapeake Bay retriever puppies.

Vitamin D receptor is essential for hair growth signaling.

Vitamin D receptor is crucial for starting hair growth after birth.

The gene SDR5A1 is found in scalp hair of both hirsute and normal individuals, but it does not explain differences in hair growth.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

A baby boy with 13q deletion syndrome had eye cancer, a woman's vision improved after stopping a breast cancer drug, a man developed cataracts from using Propecia, and a rare skin disorder called Lipoid Proteinosis was discussed. Also, a tool called OCT is useful for diagnosing macular diseases.

Certain hormone levels can help diagnose P450 oxidoreductase deficiency.

5alpha-reductase 2 is crucial for proper male brain development and sexual differentiation.

No treatment is clearly better for central serous chorioretinopathy, and more research is needed.