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A new genetic tool improves the study of hair growth and potential hair disorder treatments.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

The Kras mutation changes normal cell signals, leading to disrupted tissue structure and potential cancer.

Loss of keratin K2 causes skin problems and inflammation.

Mutations in β1 integrins cause embryonic death but have milder effects on skin.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

Targeting the TRPV4 channel may help treat intestinal diseases.

K15 and Id3 are important in hair follicle regeneration, with K15 increasing in early stages and Id3 responding later.

KAP6 genes are conserved across species and active in hair follicles.

Deleting MED1 in skin cells causes hair loss and skin changes.

Phospholipids help plant proteins move by regulating receptor interactions.

GTL1 is needed to control root hair growth and prevent problems when there are too many nutrients.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Calcium reduces involucrin in rat hair bulbs but doesn't affect filaggrin and Kdap.

PBX1 helps hair stem cells grow and change by turning on certain cell signals and preventing cell death, which may be useful for hair regrowth treatments.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

PRX01, PRX44, and PRX73 are essential for root hair growth in Arabidopsis thaliana.

Changes in KRT17 gene activity linked to wool production in Angora rabbits.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

Blocking TRPV3 may help treat itch in dry skin conditions.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

The study identified and characterized new keratin genes linked to hair follicles and epithelial tissues.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A specific gene mutation causes different hair defects in Indian monilethrix families.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

The STIM1 R304W mutation in mice leads to bone changes and teeth hair growth.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

TRα and CRABPII genes change their activity levels during goat fetal skin development.