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Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

RXRα is crucial for proper immune response and links diet to immune function.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Certain interactions help prepare the androgen receptor for pairing and activation, which is important for its role in development and disease.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

The Tru9I variant in the VDR gene may influence alopecia areata risk and vitamin D levels.

Certain VDR gene changes can affect melanoma risk.

Vitamin D receptor and hairless protein are essential for hair growth.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Vitamin D receptor gene variations are not linked to alopecia areata.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The vitamin D receptor can work without its usual activating molecule.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

Vitamin D receptor is crucial for bone health and mineral metabolism.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

RXR agonists may promote hair growth in humans.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Vitamin D receptor helps prevent skin cancer and supports skin health.

Non-liganded Vitamin D Receptor is crucial for healthy skin and hair.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

SRD5A1 is crucial in advanced prostate cancer, and blocking both SRD5A1 and SRD5A2 is more effective than targeting SRD5A2 alone.