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Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

TSC22D genes are key in metabolic diseases and cancer, offering potential as treatment targets.

The Agouti gene influences pigmentation and may have a developmental role in deer mice.

Soluble CD83 speeds up wound healing and reduces scarring.

Trichothiodystrophy hair is structurally abnormal with protein and organization issues.

MDSC-Exo can treat autoimmune alopecia areata and promote hair regrowth in mice.

Targeting ornithine decarboxylase can help prevent skin cancer.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

Removing SIX1 in fat cells reduces skin fibrosis.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Cells that move well may improve hair loss treatments by entering hair follicles.

Epidermis and dermis cells together can regenerate hair follicles.

Different types of resting melanocyte stem cells have unique characteristics and vary in their potential to become other cells.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

A new ARID1B gene variation causes Coffin-Siris syndrome 1 and early high myopia in a Chinese family.

Patients with Shwachman-Diamond syndrome often get misdiagnosed due to a wide range of symptoms, including immune system problems and bone abnormalities.

Increasing SSAT makes skin more prone to cancer.

Minoxidil helps restore thymus size in 22q11.2 deletion syndrome.

The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.

The method effectively induces skin cancer in mice for studying tumor development.

Potential therapeutic targets for scarring hair loss are identified.

The method successfully isolates hair follicle stem cells from mice for research.

Desmoglein 3 is important for keeping hair follicle stem cells inactive and maintaining their special properties.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.