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Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

The curly mutation in SELH/Bc mice affects hair and may help study human genetic disorders.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Lack of functional CYLD in mice leads to early aging and cancer.

The improved genome of the African spiny mouse will help understand its tissue regeneration abilities.

Researchers developed a mouse model that successfully mimics the bladder damage seen in humans after radiation therapy.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

C57BL/6 mice and SD rats have different sweat gland and hair follicle patterns, useful for skin research.

Adult spiny mice recover better from heart attacks than common lab mice.

Deleting part of a gene in mice causes wavy hair and high pup loss.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

Deleting the CDSN gene causes severe skin and hair problems, leading to death.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

TGFα gene mutation in mice causes abnormal skin, wavy hair, curly whiskers, and sometimes eye inflammation.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

Mice with the ab gene have abnormal sebaceous gland development, affecting sebum production.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.