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A genetic mutation in the SGK3 gene causes hairlessness in Scottish Deerhounds and may relate to human hair loss.

Multiple pregnancies prevent skin disease but worsen kidney disease in certain mice.

Mice with autoimmune hair loss showed signs of heart problems.

Touch sensitivity in mouse skin decreases during hair growth due to changes in touch receptors.

The PP2A-B55α protein is essential for brain and skin development in embryos.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Diabetic wounds heal slower than healthy wounds.

Removing hair from mice without reproductive glands led to grey hair, possibly helping to understand greying in aging.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Sonic hedgehog signaling is crucial for hair growth and maintaining hair follicle identity.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Overexpressing thrombospondin-1 in mice skin prevents UVB-induced skin damage.

Overexpressing the human H-ferritin gene in mice causes mild growth delay and temporary hair loss.

A new mouse model helps understand and find treatments for alopecia areata.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

ASH2L is essential for skin and hair development.

Schimmelpenning Syndrome requires careful evaluation and tailored treatment for skin, eye, and developmental issues.

K16 can partially replace K14 but causes hair loss and skin issues.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

High ODC and low K1 and K10 may indicate early skin tumors in mice.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Lhx2 is essential for effective Sonic Hedgehog signaling in early retinal development.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Mesenchymal MEIS2 is essential for whisker development without needing sensory nerves.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

PTHrP is important for bone formation and may be targeted for osteoporosis treatment and longevity therapies.