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Removing a specific gene in certain skin cells causes hair loss in mice by disrupting hair follicle development.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Radish saponin extract may help grow hair in balding mice.

SCD1 is important for hair growth by keeping the connection in skin cells where hair stem cells live stable.

TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

BMP2 needs periosteal tissue to help regenerate mouse middle finger bones within a specific time.

Deleting MPZL3 increases skin oil production and reduces body fat.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Certain types of mucopolysaccharidoses cause significant hair abnormalities.

Microneedle therapy combined with Hwangryeonhaedoktang solution improved hair growth in mice better than microneedle therapy alone.

Hypospadiac rats showed abnormal genital development, useful for studying human hypospadias.

High Demodex mite density may be linked to sebaceous hyperplasia.

A new gene mutation causes a rare type of hair loss.

Suppressing ODC activity reduces tumor growth in hair follicles.

Lack of TrkC receptor delays hair follicle development.

Oncogenic melanocyte stem cells can develop into melanoma similar to human cases.

Overexpression of ALK2 in hair follicles disrupts skin development and slows wound healing.

TCDD changes skin cell growth and keratin production in mice.

Blocking mTORC1 reduces skin tumor growth in mice.

Diet can prevent bone issues but not hair loss in mice lacking vitamin D receptors.

Superoxide dismutase (SOD) can prevent hair graying in mice.

Mutations in the TSPEAR gene cause a new form of ectodermal dysplasia affecting hair and tooth development.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Blocking α-toxin helps heal Staphylococcus aureus-infected wounds faster in both normal and diabetic mice, with different effects on their immune responses.

Mutations in NIPAL4 cause skin issues by disrupting lipid layers, but some improvement is seen with topical treatment.

Iron supplements may reverse premature graying in iron-deficient individuals; ingrown nails are common in diabetics with certain risk factors; topical finasteride may reduce scalp DHT as effectively as oral finasteride; monilethrix treatment is challenging but some medications can help.

Notch signaling stops Merkel cell development, and hair follicles help maintain them after skin injury.

Pincer nails are rare in lupus patients and may be managed conservatively.

A gene deletion in DSG4 causes sparse hair in some Pakistani families.