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Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

A new tool accurately identifies human cornea cell states and key factors.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

TMPRSS2 is crucial for COVID-19 infection and is a potential target for treatment.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

Hair follicle stem cell exosomes help nerve regeneration.

RNA delivery is best for in-body use, while RNP delivery is good for outside-body use. Both methods are expected to greatly impact future treatments.

BMI1 is essential for preventing hair greying and maintaining hair color.

HMG-CoA reductase is crucial for skin wound healing by regulating keratinocyte growth and blood vessel formation.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Acute stress increases Y1 receptor gene expression in certain brain areas, but repeated stress does not.

A specific RNA increases hair stem cell growth and skin healing by affecting a protein through interaction with a microRNA.

hHb1, hHb3, and hHb6 mRNAs start expressing at the same time in hair follicles.

Variants in the CCDC47 gene are linked to trichohepatoneurodevelopmental syndrome.

Hair follicle stem cell exosomes help nerve regeneration.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Nestin-expressing hair follicle cells may be useful for nerve repair and regeneration.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

LONRF1 is important for oxidative damage response and tissue remodeling during wound healing.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Defects in certain proteins cause major heart abnormalities during early development.

Using special RNA to target a mutant gene fixed hair problems in mice.

The KRTAP11-1 gene promoter is crucial for specific expression in sheep wool cortex.

A special treatment speeds up chronic wound healing by fixing cell energy issues and reversing aging in cells.