Search

everythinglearnresearchcommunity

for

Search:↓

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

The pituitary gland functions normally even after losing most SOX2+ stem cells.

Ovol2 is important for proper skin healing and hair growth.

Overexpression of SSAT in mice causes hair loss, liver damage, and sensitivity to polyamine analogues.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

Par3 protein is essential for skin cell balance and stability.

GFP transgenic mice help study cell origins in skin grafts.

Sensory neuron remodeling and Merkel-cell changes happen independently during skin maintenance.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Deleting MED1 in skin cells causes hair loss and skin changes.

A new genetic mutation in the TRPS1 gene causes Trichorhinophalangeal Syndrome, leading to specific hair, dental, and bone issues.

Tanning ability is linked to specific DNA changes in skin genes.

African spiny mice can regenerate skin, hair, and cartilage, but not muscle, and their unique abilities could be useful for regenerative medicine.

Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

Lack of KSR1 stops certain skin tumors in mice.

PBX1 reduces aging and cell death in stem cells by boosting SIRT1 and lowering PARP1.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

Mouse hair follicle stem cells have a flexible chromatin state that supports skin health and hair growth.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

The study found that p63 needs signals from morphogens to help skin cells differentiate properly.

The study found that aging hair follicle stem cells change behavior, leading to hair loss, but drugs targeting these cells may help restore hair growth.

The document concludes that stem cell therapies lack solid proof of effectiveness, except for blood system treatments, and criticizes the ethical issues and commercial exploitation in the field.

The symposium concluded that understanding how different species repair tissue and how this changes with age can help advance regenerative medicine.

Adult tissue stem cells can adapt and switch roles to help repair and maintain the body.

A gene mutation causes curly hair and hair loss in rats.

Understanding snoRNA regulation may help slow skin aging.