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A specific RNA increases hair stem cell growth and skin healing by affecting a protein through interaction with a microRNA.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

The HCR gene contributes to psoriasis risk.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Fibroblast bioelectric signaling can promote hair growth and may help treat hair loss.

Mefloquine, an antimalarial drug, is effective in killing melanoma cells resistant to other treatments by causing lethal stress in the cells.

Light can turn on hair growth cells through a nerve path starting in the eyes.

A mutation in the Sgkl gene causes defective hair growth in mice.

TPA is about 50 times more effective at promoting tumors than MZ.

Cyclosporin A and FK506 can start new hair growth in mice, but ascomycin and rapamycin cannot.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.

HPV8 E6 gene causes growth of certain skin stem cells.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

PPARγ signaling modulation can protect hair follicle stem cells from chemotherapy-induced damage.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Shikonin, a small molecule, speeds up burn wound healing and hair growth by activating the PI3K/Akt pathway.

TPA promotes hair growth by increasing stem cell activity and activating specific cell signals.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

CRH can cause hair loss by promoting cell death in hair growth cells.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Senescent fibroblasts can help start tumors in normal skin cells.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Blocking RANK signaling might help treat metastatic melanoma, but more research is needed.

Cyclosporin A promotes hair cell growth and affects protein kinase C levels.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Poncirin is a promising inhibitor of Janus Kinase 3, potentially better than tofacitinib.

Hair follicle samples effectively show how well the drug MK-0752 targets and engages with the Notch pathway.