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Glucocorticoids and thyroid hormones together are essential for fetal fat development.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

KLHL24-mutant stem cells help understand skin and heart disease.

Significant progress was made in understanding PXE, but effective treatments are still needed.

Epidermal β-catenin activation changes the dermis by signaling different fibroblast types.

Basonuclin is crucial for hair follicle development and cycling in mice.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Keratin 17 is crucial for normal hair growth by regulating hair cycle transitions with TNFα.

RXR and RAR proteins in skin may help with cell growth, hair growth, and gland function.

Blocking Hedgehog signaling offers new treatment options for advanced basal cell carcinoma.

Basement membrane changes are crucial for hair follicle development.

The E2 protein affects gene activity in hair follicles of mice.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

The fuzzy gene is crucial for controlling hair growth cycles.

TPA promotes hair growth by increasing stem cell activity and activating specific cell signals.

Exosomal miR-222-3p reduces melanin production in rabbits by targeting the SOX10 gene.

miR-200a reduces goose fibroblast growth by targeting PITX2 in the Wnt pathway.

A new PAX9 gene mutation causes missing teeth and hair problems, but not skin or nail issues.

Basonuclin may help control ribosomal RNA gene activity in skin cells.

BMP signaling prevents hair growth by stopping stem cell activation.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

GKLF/KLF4 and Sp1 control Keratin 19 gene activity, influencing cancer-related changes.

Hox proteins help maintain keratinocyte identity by regulating miRNA expression.

Notch/RBP-J signaling is crucial for proper placement and timing of melanocyte development in hair follicles.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Noggin promotes skin tumors by activating certain cell signaling pathways.

Temporary activation of β-catenin can create new hair follicles, but ongoing activation is needed to keep hair follicle tumors.