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CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Blocking casein kinase 1 in skin cells can help melanocyte precursors move better, potentially helping with conditions like vitiligo or gray hair.

αCT1 improves scar appearance by changing early collagen structure.

Touch dome keratinocytes in adult skin have traits of different skin cell types.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

CTHRC1 helps hair grow back, and plantar dermis mixture boosts it.

T cells with memory features grow in number and gather around hair follicles when there are not enough immune cells.

Cavity macrophages gather on organ surfaces but don't really invade or help repair the organs after injury.

Cellularity can estimate mesenchymal stem cell concentration in bone marrow, aiding regenerative medicine.

Long-standing benign tumors can become cancerous, especially in people with weakened immune systems.

TCDD changes skin cell growth and keratin production in mice.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Claudin-1 is crucial for maintaining skin barrier and preventing inflammation.

The HATMSC1 cell line from fat tissue can produce helpful factors for regenerative and immune therapies.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

People with myotonic dystrophy type 1 tend to develop basal cell carcinoma at a younger age but not more frequently than others.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Adipose-derived stem cell-conditioned medium can reduce melanoma cell growth and spread.

Clonally expanded CD8+ T cells cause alopecia areata.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Inactive stem cells in hair follicles and muscles can avoid detection by the immune system.

Mcl-1 can activate Wnt signaling in skin cells, promoting growth and possibly cancer.