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Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

RXRα is crucial for hair growth and skin cell function.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Gene therapy in mice increased lifespan and improved health without causing cancer.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Researchers made a mouse model with curly hair and hair loss by editing a gene.

The Itpr3 gene causes a specific hair pattern in mice.

IL-18 signaling helps mature Tregs move into the thymus.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Researchers created immortal human skin cells with constant testosterone receptor activity to study hair loss and test treatments.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

Overexpressing the human H-ferritin gene in mice causes mild growth delay and temporary hair loss.

The E2 protein affects gene activity in hair follicles of mice.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Lack of KSR1 stops certain skin tumors in mice.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

The retinoid receptor antagonist effectively disrupts vitamin A-related development in embryos.