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Blocking a specific enzyme in male rat fetuses leads to the development of nipples and feminized genitalia.

A gene variant in KRT71 causes the curly fur in Selkirk Rex cats.

The Foxn1 gene is essential for normal nail and hair development.

The human K5 promoter controls specific gene expression in skin cells, with key regulatory elements near the TATA box.

Truncated LTBP-1 disrupts TGF-beta signaling, affecting hair growth.

Lack of TG2 increases fat storage and lowers cell cleanup in skin oil cells.

The technology can create transgenic cashmere goats with improved wool quality.

Inhibiting TYK2 can restore hair growth in alopecia areata.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Two mouse mutations cause similar hair loss despite different skin changes.

Blocking TRPV3 may help treat itch in dry skin conditions.

DKK4 can be used to improve wool quality in Zhexi Angora rabbits.

Replacing defective mesenchymal cells with normal ones fixes thymic growth issues in 22q11.2DS mouse models.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The Kras mutation changes normal cell signals, leading to disrupted tissue structure and potential cancer.

Male and female mice handle stress differently.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Removing the ROBO4 gene in mice reduces skin inflammation and hair loss by affecting certain inflammation pathways and gene expression.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

Skin cancer often starts from Lgr5+ progenitor cells.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Ornithine decarboxylase is crucial for hair growth regulation in mice.

cDermo-1 causes dense skin, feathers, and scales in chickens.

The QuantAnts machines can find cancer markers and create CRISPR targets for them.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

Zebrafish with mutations similar to human Cantú Syndrome have heart cells with altered channel properties, making them a good model to study the condition.