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Severe CCCA may be biologically and clinically different from milder forms.

Hair evaluation is crucial for early diagnosis and management of ARCI, as hair loss often indicates severity.

The proteins transthyretin and megalin are more present in the growth phase of hair, suggesting they might affect hair health and growth.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Lgr6+ cells are important for tissue repair and could be a target for treating cancer and other diseases.

Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Tissue-engineered skin substitutes can model junctional epidermolysis bullosa and may help develop gene therapy.

The NUDT15 R139C variant causes thiopurine-induced leukocytopenia through a different mechanism than previously thought in Japanese patients with inflammatory bowel disease.

The E2 protein affects gene activity in hair follicles of mice.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

Researchers converted human embryonic stem cells into trophoblast stem cells using specific transcription factors.

Syndecan-1 is essential for maintaining skin fat and preventing cold stress.

Rituximab may be an effective treatment for Cronkhite-Canada syndrome with kidney disease.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Newer retinoid drugs are effective for skin conditions but have significant side effects.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

A new topical treatment may prevent hair loss from cancer therapy by adjusting cell death processes in hair follicles.

Vitamin D receptor is crucial for skin wound healing.

Hairless protein can block vitamin D activation in skin cells.

TTD hair is brittle due to fewer sulfur amino acids and unstable disulfide bonds.

LncRNA MTC boosts growth of goat skin cells, improving cashmere quality.

CDH3-related disease causes worsening eye and hair issues.

Newly designed proteins can effectively degrade specific proteins in cells, offering a potential new therapy method.

miR-150 helps blood vessel cells develop and speeds up blood clot healing.

Cantú syndrome is caused by mutations in the ABCC9 gene.

Vitamin D3 and its receptor are important for hair growth, and understanding receptors could help treat hair graying and skin cancer.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

A new genetic variant causes BRESHECK syndrome by disrupting cell growth and stress response.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.