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The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

KY19382 speeds up wound healing by activating a specific cell signaling pathway.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

Stat3 affects skin cancer development by altering keratinocyte stem cell behavior.

Increasing Wnt5a in mice skin delays hair growth but doesn't stop it.

E-cadherin is important for cell movement in electric fields, and the new tracking method works well.

Meis1 is crucial for skin health and tumor development.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Fibroblast and immune cell interactions affect tissue repair and fibrosis.

Connexin-26 gene mutations may increase cancer risk in KID syndrome patients.

lncRNA MTC affects protein levels in goat skin cells, impacting hair growth.

Keratin-associated proteins may have roles in various mouse tissues, not just hair.

Overexpression of ΔNLef1 in mouse skin leads to hair loss, cysts, and skin tumors.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

Blocking a specific receptor slows down hair loss in mice.

TGF-β receptor 1 helps deer antlers grow by aiding skin and cartilage cell growth.

Necl2 affects skin cell behavior and slows wound healing.

Upadacitinib may effectively treat twenty-nail dystrophy without causing pain.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Fetal wound healing changes with development, affecting inflammation and collagen, which may influence scarring.

The Foxn1(-/-) phenotype disrupts hair growth and affects skin stem cells.

Desmoglein 3 organization in cell connections changes without calcium, affecting cell adhesion.

Patched1 helps prevent tumors by controlling cell growth.

Wnt signaling can improve skin healing by promoting epithelial growth.

Epidermal Wnt/β-catenin signaling controls fat cell formation and hair growth.